On August 17, 2026, Shenzhen Bay Laboratory Institute of Brain Disorders and the Kunming Institute of Zoology (KIZ) jointly held an academic symposium in Kunming. Over a dozen experts and scholars from both institutions gathered to discuss non-human primate disease models, neurological disease mechanisms, and brain imaging technologies.

The Shenzhen Bay Laboratory Institute of Brain Disorders focuses on autism, chronic pain, depression, sleep disorders, schizophrenia, and Alzheimer's disease, aiming to better understand, diagnose, and treat neurological and psychiatric conditions. KIZ, as a comprehensive research institution in biodiversity evolution, conservation, and sustainable utilization, hosts the National Key Laboratory of Genetic Evolution and Animal Models, the National Major Science and Technology Infrastructure for Phenotypic and Genetic Studies of Model Animals (Primate Facility), and the National Non-Human Primate Experimental Animal Resource Bank.

The symposium opened with a welcome address from Prof. Gan Wenbiao, Director of the Shenzhen Bay Laboratory Institute of Brain Disorders. KIZ Deputy Director Prof. Wu Dongdong, also Director of the National Key Laboratory of Genetic Evolution and Animal Models and initiator of the "Primate Genome Project," delivered the first presentation on non-human primate evolutionary genetics and disease models.

KIZ researchers presented their latest findings: Prof. Kong Qingpeng shared research on endogenous anti-aging factors in long-lived populations; Prof. Zhang Dengfeng introduced Alzheimer's disease models using macaques and brain organoids; Prof. Hu Xintian reported on clinical standardized evaluation and large-scale modeling systems for major brain disease macaque models based on human-monkey disease homology, noting that his team was the first to report spontaneous macaque models of Parkinson's and Alzheimer's diseases; and Prof. Sheng Nengyin discussed synaptic pathogenic mechanisms in intellectual disability.

From the Shenzhen Bay Laboratory side, researchers presented on transcriptional dynamics in human brain evolution, synaptic pathology mechanisms, primate models for depression and stroke treatment, Golgi apparatus in disease, glycolysis-mitochondrial balance, APOE4 and innate immunity, neuroinflammation, focused ultrasound neuromodulation, and brain PET imaging technology.

The symposium assembled multidisciplinary expertise spanning neuroscience, genetics, disease model construction, and brain imaging, laying a solid foundation for deeper collaboration. The two institutions will use this symposium as a starting point to advance non-human primate model innovation in major brain disease research and promote clinical translation of brain science discoveries.